Table S9 Effect of genome-wide significant or replicating variants on glucose, anthropometric and lipid traits. Launer, Michael A. SNPs are plotted by position on chromosome against association with CT hepatic steatosis —log10 p-value. Less is reported on the role of ESR1 in males. To understand the transcriptional control of overall lipid metabolism, we determined the expression of fatty acid oxidative genes. Articles include original research papers, reviews or mini-reviews, and short research communications. Folch, J. Perry, R. Absher, Najaf Amin, Anna L.
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Alcohol Related Liver Disease. A baker claims she was almost made infertile after the contraceptive pill left her with a six-inch tumour in her liver. Tasha Luck, 25, was.
O'Donnell, Cameron D. Acknowledgments We would like to thank Dr. However, the variants with the lowest p-values of association with CT hepatic steatosis near LYPLAL1 and PPP1R3B fall in non-coding regions and thus for these as well as the other three loci above we tested whether they have effects on the expression of nearby genes in liver and adipose tissue from a sample of bariatric surgery patients  See Table S Mice were fasted overnight, and serum was collected by facial vein puncture.
Visscher, Themistocles L.
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University of Exeter Medical School, RILD. Nonalcoholic fatty liver disease (NAFLD) clusters in families, but the only Citation: Speliotes EK, Yerges-Armstrong LM, Wu J, Hernaez R, Kim LJ, M.
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Karim Rumi Molecular and Cellular Endocrinology Lundholm et al. Abstract Impaired estrogens action is associated with features of the metabolic syndrome in animal models and humans. One possible mechanism for contributing to both increased hepatic lipogenesis and gluconeogenesis is impaired insulin signaling. We could not assess whether the recently reported NAFLD associated variants near APOC3  associate with CT hepatic steatosis as they were not genotyped on the Affymetrix or Illumina platforms used by our studies and these variants do not have proxies that we could use in HapMap to impute them.
This result suggested that other receptors might be involved in E 2 mediated Fas and Acc1 transcriptional activation. Amino acid-dependent activation of liver estrogen receptor alpha integrates metabolic and reproductive functions via IGF
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|The experiments were performed 1—3 weeks after virus injection.
D Quantification of O2 consumption. Randall, Sailaja Vedantam, Thomas W. Therefore, an understanding of the mechanisms underlying gonadal steroid regulation of glucose and lipid metabolism is required.
In the first stage, 1, subjects were chosen, equally distributed between the highest and lowest quartiles of age- and sex-adjusted values for coronary artery calcification, assessed by CT scan.
Andrews. as subordinate had higher copper uptake in their gills and liver, which was correlated with a higher uptake of sodium. Andrew Joseph Muir's profile, publications, research topics, and co-authors.
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Hepatitis C; Primary sclerosing cholangitis; Cirrhosis; Liver Transplantation. liver transplantation.
Kimberly A. Powers. As the new liver was reinfected, production increased and viral load rose to a new steady state.
Cancer ; 11 3 : Article PubMed Google Scholar The epidemiology of nonalcoholic fatty liver disease in adults.
Home About License Contact. Monty Krieger for critically reviewing the manuscript. Efstathiadou, Z. Kim10 Cameron D.
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|CT images of the chest were used to measure liver attenuation corresponding to superior aspects of the right and medial lobes or hepatic segments 4a, 7 and 8 using the Couinaud system.
Berndt, Keri L. Therefore, an understanding of the mechanisms underlying gonadal steroid regulation of glucose and lipid metabolism is required. Table 1 Characterization of family data for heritability estimation. National Center for Biotechnology InformationU.
Nonalcoholic fatty liver disease: a systematic review. Amino acid-dependent activation of liver estrogen receptor alpha integrates metabolic and reproductive functions via IGF